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http://projects.csail.mit.edu/courseware/?term=the-giving-tree-essay the giving tree essay T is nomenclature has allen out o avor as basilar artery caliber is not altered during an attack. Symptoms as de ned by the international headache society include1. Dysarthria vertigo innitus diplopia hypoacusis ataxia decreased level o consciousness what are the di erent types kn wn x amilial hemiplegic migraine (fhm)1,8 ?. Fhm1. De ned as a mutation in the cacna1a gene, coding or a p/q voltage-gated calcium channel, on chromosome 19 with the accompanying clinical syndrome and appropriate amily history in a rst- or second-degree relative. T is mutation is responsible or around 50% o fhm cases. Cerebellar ataxia, either progressive orepisodic in nature, is a common accompaniment. Minor head trauma can trigger an attack, and brainstem signs such as coma can occur, o en prompting aggressive treatment or epilepsy or encephalitis in patients. Fhm2. T ere are mutations in the atp1a2 gene, coding or a k/na-a pase, on chromosome 1. T is accounts or around 20% o fhm cases. An epilepsy phenotype may occur. T is mutation may also be responsible or alternating hemiplegia o childhood.

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http://cs.gmu.edu/~xzhou10/semester/thesis-and-hypothesis.html thesis and hypothesis »» lamivudine lamivudine (epivir-hbv) is an oral synthetic cytosine nucleoside analog with antiviral effects viagra pentru femei gold max against hiv and hbv. Lamivudine is effective in suppressing hbv replication, normalizing alt levels, and improving liver histology. Patients may have a similar or a superior response in achieving these endpoints when compared with interferon or pegylated interferon. Prolonged lamivudine therapy (up to 5 years) may be needed to sustain seroconversion, but this leads to lamivudine resistance as high as 60% to 70% at 5 years. 30 due to the high rate of resistance, lamivudine is no longer recommended as first-line therapy for chb. 30 the adult dose of lamivudine is 100 mg orally once daily for chb without hiv coinfection. Lamivudine 3 mg/kg once daily up to a maximum dose of 100 mg is approved for pediatric patients (2–17 years of age).

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http://cs.gmu.edu/~xzhou10/semester/thesis-binding-drayton-park.html thesis binding drayton park 1. The pathophysiology of ttts is not completely understood, but placental vascular anastomoses, unequal placental sharing, and abnormal umbilical cord insertions are all necessary for tits to occur. Eighty-five percent of monochorionic placentas have vascular connections that include superficial arterial-to-arterial (aa) and venous-to-venous (vv) anastomoses that have bidirectional how and deep interfetal artery-to-vein (av) communications with unidirectional how located in the placental cotyledons that are supplied by one fetus and drained by the other. The number and type of anastomoses impact whether the exchange of blood between the twins is balanced or unbalanced. Ttts results when there is limited bidirectional flow through aa or vv connections. Aa connections are thought to be protective, associated with a ninefold reduction in the risk of developing chronic tits, while av anastomoses with unidirectional flow lead to shunting of blood from one twin to the other and are associated with worse perinatal outcome. Ten percent to 20% of monochorionic placentas have sufficient circulatory imbalance to produce tits. One fetus (the donor) slowly pumps blood into the cotwin's circulation (the recipient). Complications in the donor include anemia, hypovolemia and resultant activation of the renin-angiotensin-aldosterone system, growth restriction, brain ischemic lesions, renal hypoperfusion and insufficiency, oligohydramnios ("stuck twin''), lung hypoplasia, limb deformation, and high risk for fetal demise. Complications in the recipient include polycythemia, thrombosis, cerebral emboli, disseminated intravascular coagulation (dic), polyhydramnios, progressive cardiomyopathy due to volume 130 i multiple births overload, and fetal hydrops. Newer evidence suggests that the pathophysiology oftits involves changes in the renin-angiotensin system and increased levels of human brain natriuretic peptide (hbnp), atrial natriuretic peptide (anp), and endothelin-1. Vasoactive mediators produced in the donor are shunted to the recipient resulting in hypertension and contributing to the development of hypertensive cardiomyopathy and hypertensive microangiopathy. 2. Diagnosis is usually made between 17 and 26 weeks' gestation, but the process may occur as early as 13 weeks.

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https://graduate.uofk.edu/user/diploma.php?sep=biology-assignment-help biology assignment help Sleep deprivation may also be used •• video-electroencephalogram (veeg) epilepsy are very small. In these cases, the only necessary pharmacotherapy is to correct the underlying problem and possibly use an aed short-term. Risk factors for repeated seizures in patients without an underlying disorder include:21 •• structural cns lesion •• abnormal eeg •• partial seizure type •• positive family history •• postictal motor paralysis if no risk factors are present, the risk of another seizure is 10% to 15%. However, if two or more risk factors are present, the risk of another seizure is 100%. If the estimated risk is over 70%, an epilepsy diagnosis is plausible. When sufficient evidence is available to determine the patient has seizures and is at risk for another seizure, pharmacotherapy is usually started (figure 31–2). The patient should be in agreement with the plan, be willing to take the medication, and be able to monitor seizure frequency and adverse drug effects. Mechanisms of action, effectiveness for specific seizure types, common adverse effects, and potential drug interactions are key elements in selecting a medication. Other patient factors such as gender, concomitant drugs, age, economic factors, and lifestyle also need to be considered. Nonpharmacologic therapy many of the tests are done to rule out other causes of seizures (eg, infection, electrolyte imbalance), and in most patients will be normal. Often the eeg appears normal between seizures. 20 sleep deprivation, photic stimulation, prolonged (greater than 20 minutes) eeg recording, and 24-hour eeg monitoring with video correlation can be done to capture seizure or seizure-like activity on the eeg.

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