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algerbra homework help 7. Infections. Bacteremia, hepatitis, cytomegalovirus (cmv), human immunodeficiency virus (hn) (acquired immune deficiency syndrome [aids]), west nile virus, and malaria (see chaps. 48 and 49). 8. Hemolysis. Hemoglobinemia, hemoglobinuria, and hyperkalemia caused by overheating of the blood have been reported. Massive hemolysis, intravascular sickling, and death have occurred from the use of hemoglobin sickle cell (sc) donor blood. 332 i neonatal hyperbilirubinemia 9.

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http://cs.gmu.edu/~xzhou10/semester/senior-thesis-books.html senior thesis books Physical examination once seizures are controlled, a neurologic exam should evaluate the level of consciousness (coma, lethargy, or somnolence), motor function and reflexes (rhythmic contractions, rigidity, spasms, or posturing), and pupillary response. A physical exam can identify secondary injuries. Clinical symptoms patients with se usually present with generalized, convulsive, tonic-clonic seizures. They may also be hypertensive, tachycardic, febrile, and diaphoretic which resolve after seizure termination. A loss of bowel or bladder function, respiratory compromise, and nystagmus may also be observed. When seizure activity symptoms •• impaired consciousness ranging from lethargy to coma •• disorientation after cessation of gcse •• pain from associated injuries (eg, tongue lacerations, dislocated shoulder, head trauma, facial trauma) signs phase i. Phase ii (greater than 30 minutes of se). Generalized convulsions hypertension, tachycardia fever and sweating muscle contractions, spasms respiratory compromise incontinence respiratory failure with pulmonary edema cardiac failure (arrhythmias, shock) hypotension hyperthermia rhabdomyolysis and multiorgan failure laboratory tests •• hyperglycemia (phase i) and hypoglycemia (phase ii) can occur •• hyponatremia, hypernatremia, hyperkalemia, hypocalcemia, hypomagnesemia, and hypoglycemia can cause se •• the white blood cell (wbc) count may slightly increase •• abnormal arterial blood gases (abgs) due to hypoxia and respiratory or metabolic acidosis •• elevated serum creatinine will be present in renal failure patients •• myoglobinuria can occur in patients with continuous seizures diagnostic tests eeg to confirm seizure activity ct or mri may reveal mass lesions or hemorrhage exceeds 30 to 60 minutes, muscle contractions may no longer be visible, necessitating an eeg to diagnosis se. Twitching of the face, hands, or feet may be seen in these comatose patients with prolonged seizures. Laboratory parameters it is important to obtain a serum chemistry profile to help identify possible causes of se, such as hypoglycemia, hyponatremia or hypernatremia, hypomagnesemia, and hypocalcemia. Patients with renal and liver failure are at high risk as well. In febrile patients with elevated white blood count (wbc) counts, active infections should be considered and treated appropriately. Cultures from the blood, cerebrospinal fluid (csf), respiratory tract, and urine should be collected once seizures are controlled. Computed tomography (ct) or magnetic resonance imaging (mri) can rule out cns abscesses, bleeding, or tumors, all of which may be a source of seizures.

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