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http://cs.gmu.edu/~xzhou10/semester/difference-between-thesis-and-journal-article.html difference between thesis and journal article 51). Iii. This is an opportune time to explore aids and hepatitis c risks with the mother. The discussion may include a recommendation for hiv and/or hepatitis c testing for which specific patient consent is required (see chap. 48). A drug-addicted mother is at increased risk for other diseases such as sexually transmitted diseases, tuberculosis, hepatitis b and c, and aids, especially if she involves hersdf in intravenous drug use or prostitution. Approximatdy 30% of pregnant intravenous drug users are seropositive for hiv (see chap. 48). Ill. Narcotic exposure in pregnancy. Methadone, heroin, and prescribed narcotics are the most common reasons for withdrawal seen in our nurseries. Prescribed narcotics, such as morphine, fentanyl, percocet, and dilaudid, are given in pregnancy for management of chronic pain despite their dependence potential. There has also been a rise in the use of oxycontin in this country since the early 2000s when reports surfaced about the abuse of this opioid. Both its illicit as well as legal use is seen in pregnant women. Oxycontin is the extended-release form of oxycodone, an opioid twice as potent as oral morphine. It was originally thought that the extendedrdease properties would lower the abuse potential.

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http://www.cs.odu.edu/~iat/papers/?autumn=help-with-chemical-engineering-homework help with chemical engineering homework Who. Int/cancer/palliative/painladder/en/. 36. Zhang w, moskowitz rw, nuki g, et al. Oarsi recommendations for the management of hip and knee osteoarthritis, part ii. Oarsi evidence-based, expert consensus guidelines. Osteoarthritis cartilage. 2008;16:137–162. 37. Kurth t, glynn rj, walker am, et al. Inhibition of clinical benefits of aspirin on first myocardial infarction by nonsteroidal anti-inflammatory drugs. Circulation. 2003;108:1191–1195. 38. Moore ra, derry s, mcquay hj. Cyclo-oxygenase 2-selective inhibitors and nonsteroidal anti-inflammatory drugs. Balancing gastrointestinal and cardiovascular risk. Bmc musculoskelet disord. 2007;8:73. 39. American pain society. Principles of analgesic use in the treatment of acute pain and cancer pain. 5th ed. Glenview, il. American pain society. 2003:13–41.

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pay for a research paper B. Meconium staining of cord and nails due to long-term exposure to msaf. C. A higher risk of fetal, intrapartum, or neonatal death. E. Management 1. Antepartum management a. Careful estimation of true ga, including ultrasonographic data. B. Antepartum assessments by cervical examination and monitoring of fetal well-being {see chap. 1) should be initiated between 41 and 42 weeks on at least a weekly basis. If fetal testing is not reassuring, delivery is usually indicated. In most instances, a patient is a candidate for induction of labor if the pregnancy is at >41 weeks of gestation and the condition of the cervix is favorable. 2. Intrapartum management involves use of fetal monitoring and preparation for possible perinatal depression and meconium aspiration. 3. Postpartum management a. Evaluation for other conditions.

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http://manila.lpu.edu.ph/about.php?test=essay-formats essay formats Ii. Epidemiologic associations. Pphn occurs at a rate of 1 to 2 per 1,000 live births and is most common among full-term and postterm infants. Perinatal risk factors reported in association with pphn include meconium-stained amniotic fluid and maternal conditions such as fever, anemia, and pulmonary disease. Case-control studies of risk factors for pphn suggest associations between pphn and a number of antenatal and perinatal factors, including maternal diabetes mellitus, urinary tract infection during pregnancy, selective serotonin reuptake inhibitors (ssris), aspirin, nonsteroidal anti-inflammatory drug consumption during pregnancy, and cesarean section delivery. Although mechanisms of antenatal pathogenesis remain uncertain, there are a number of perinatal and neonatal conditions that have well-established links with pphn. 435 436 i persistent pulmonary hypertension of the newborn a. Severe fetal hypoxemia ("asphyxia'') is the most common associated diagnosis. Some speculate that prolonged fetal stress and hypoxemia lead to remodeling and abnormal muscularization of pulmonary arterioles. Acute birth asphyxia also causes release of vasoconstricting humoral factors and suppression of pulmonary vasodilators, thus contributing to pulmonary vasospasm. B. Pulmonary parenchymal diseases, including surfactant deficiency, pneumonia, and aspiration syndromes, such as meconium aspiration, are also associated with increased risk of pphn. In most such cases, the pulmonary hypertension is reversible, suggesting a vasospastic contribution. However, concomitant pulmonary vascular remodeling cannot be excluded. The risk of pulmonary hypertension appears to be greater when the fetus is of more advanced gestational age, suggesting that the stage of pulmonary vascular development might play a role in susceptibility to pphn. C. Abnormalities of pulmonary development contribute structurally to pphn, either by pruning of the vascular tree, as occurs in congenital diaphragmatic hernia, potter syndrome, and other forms ofpulmonary parenchymal hypoplasia, or malalignment of pulmonary veins and arteries, as is seen in alveolar capillary dysplasia. D.

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