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http://www.cs.odu.edu/~iat/papers/?autumn=homework-help-about-facts-details homework help about facts details Circulation. 2013;129(3):E28-e292. 2. World health organization. Mortality and global health estimates. Causes o death. En leading causes o death, 2012 (by sex). 2013. Apps.Who.Int/gho/data/view.Wrapper. Mghemor cause10-2012?. Lang= en&menu= hide. Accessed july 19, 2014. 3. Yusu s, hawken s, ounpuu s, et al. E ect o potentially modi iable risk actors associated with myocardial in arction in 52 countries (the in erhear study). Casecontrol study. Lancet. 2004;364(9438):937-952. 4. Lloyd-jones dm, hong y, labarthe d, et al. De ining and setting national goals or cardiovascular health promotion and disease reduction. The american heart association's strategic impact goal through 2020 and beyond. Circulation. 2010;121(4):586-613. 5. Wilson pw, d’agostino rb, levy d, belanger am, silbershatz h, kannel wb. Prediction o coronary heart disease using risk actor categories. Circulation. 1998;97(18):1837-1847. 6. Stamler j, vaccaro o, neaton jd, wentworth d.

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http://projects.csail.mit.edu/courseware/?term=checking-essay-for-plagiarism checking essay for plagiarism Pediatr clin n am 2007;54(5):787-798. Nutrition deirdre m. Ellard and diane m. Anderson following birth, term infants rapidly adapt from a rdativdy constant intrauterine supply of nutrients to intermittent feedings of milk. Preterm infants, however, are at increased risk for potential nutritional compromise. These infants are born with limited nutrient reserves, immature metabolic pathways, and increased nutrient demands. In addition, medical and surgical conditions commonly associated with prematurity have the potential to alter nutrient requirements and complicate adequate nutrient delivery. As survival for these high-risk newborns continues to improve, current data suggest that early, aggressive nutrition intervention is advantageous. I. Growth a. Fetal body composition changes throughout gestation, with accretion of most nutrients occurring primarily in the late second and throughout the third trimester. Term infants will normally have sufficient glycogen and fat stores to meet energy requirements during the rdative starvation of the first day after birth. In contrast, preterm infants will rapidly deplete their limited nutrient reserves, becoming both hypoglycemic and catabolic unless appropriate nutritional therapy is provided. In practice, it is generally assumed that the severity of nutrient insufficiency is inversdy related to gestational age at birth and birth weight. B. Postnatal growth varies from intrauterine growth in that it begins with a period of weight loss, primarily through the loss of extracellular fluid. The typical loss of 5% to 10% of birth weight for a full-term infant may increase to as much as 15% of birth weight in infants born preterm. The nadir in weight loss usually occurs by 4 to 6 days of life, with birth weight being regained by 14 to 21 days of life in most preterm infants. Currently, there is no widely accepted measure of neonatal growth that captures both the weight loss and subsequent gain characteristic of this period. Goals in practice are to limit the degree and duration of initial weight loss in preterm infants and to facilitate regain of birth weight within 7 to 14 days oflife. C. After achieving birth weight, intrauterine growth and nutrient accretion rate data are widdy accepted as reference standards for assessing growth and nutrient requirements. Goals of 10 to 20 g/kglday weight gain (15-20 glkglday for infants < 1,500 g), approximately 1 em/week in length, and 0.5 to 1 em/week in head circumference are used. Although these goals are not initially attainable in most preterm infants, replicating growth of the fetus at the same gestational age remains an appropriate goal as recommended by the american academy of pediatrics (aap).

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